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Research Article | Open Access

E5 peptide-functionalized platinum nanozymes mediate immune microenvironment reprogramming in B-cell lymphoma

Mengjun Wang1Rong Guo2Yitong Zhao1Ying Wang1Wenxin Fu1Guancheng Wang1Mingze Lu1Hao’an Wu1 ( )Ming Ma1 ( )Yunfei Bai1 ( )Yu Zhang1 ( )
State Key Laboratory of Digital Medical Engineering, Jiangsu Key Laboratory for Biomaterials and Devices, School of Biological Science and Medical Engineering & Basic Medicine Research and Innovation Center of Ministry of Education, Zhongda hospital, Southeast University, Nanjing 211189, China
Department of Pharmacy, The First Affiliated Hospital of Zhengzhou University, Zhengzhou 450052, China
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Abstract

To address the complexity of structural design and synergistic optimization of biological effects of nanozymes in the field of oncology therapy, this study constructed a nanozymes platform (Pt@E5) with a facile and highly efficient preparation process for precision targeting of lymphoma. Pt@E5 nanozymes assemblies were directly prepared by a one-step reduction method, and the modification of E5 peptide ensured the dimensional homogeneity and stability of the platinum nanoparticles. This nano enzymatic platform combines dual enzyme activity with chemokine receptor 4 (CXCR4) targeting. It can inhibit tumor metastasis by targeting CXCR4 and thus specifically blocking the CXCR4/CXCL12 (stromal cell derived factor 1α) signaling pathway. In addition, it induces mitochondria mediated apoptosis and immunogenic cell death (ICD) in tumor cells by catalyzing the production of reactive oxygen species (ROS) using dual enzyme activity. In a mouse lymphoma model, the therapeutic results showed that Pt@E5 treatment effectively remodeled the tumor immunosuppressive microenvironment by promoting cytotoxic T cell infiltration (CTLs) and reducing regulatory T cell (Treg) levels. It also eliminated A20 cells from the bone marrow and peripheral blood, leading to significant tumor regression compared to the control group. In conclusion, this study offers a promising translational strategy for the multifaceted collaborative treatment of diffuse large B-cell lymphoma (DLBCL) based on a simple nanozyme-peptide assembly.

Graphical Abstract

This work reports a novel Pt@E5 nanozyme mediating targeted killing of B-cell lymphoma. Following tail-vein injection, Pt@E5 nanozymes actively bind to chemokine receptor 4 (CXCR4) on the surface of A20 cells, enabling targeted endocytosis. Pt@E5 nanozymes catalyze reactive oxygen species (ROS) bursts within cells through enzyme-like activity, deplete glutathione, reduce mitochondrial membrane potential, and thereby trigger apoptosis and immunogenic cell death (ICD). Additionally, Pt@E5 nanozymes eliminate A20 cells from peripheral blood circulation and block their homing to bone marrow, thereby synergistically inhibiting B-cell lymphoma progression through multiple mechanisms.

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Nano Research
Article number: 94908016

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Cite this article:
Wang M, Guo R, Zhao Y, et al. E5 peptide-functionalized platinum nanozymes mediate immune microenvironment reprogramming in B-cell lymphoma. Nano Research, 2025, 18(10): 94908016. https://doi.org/10.26599/NR.2025.94908016

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Received: 01 July 2025
Revised: 27 August 2025
Accepted: 28 August 2025
Published: 19 September 2025
© The Author(s) 2025. Published by Tsinghua University Press.

This is an open access article under the terms of the Creative Commons Attribution 4.0 International License (CC BY 4.0, https://creativecommons.org/licenses/by/4.0/).