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Rapid Communication | Open Access

Cis-regulation of FAM49A by a risk variant at 2p24 contributes to the genetic susceptibility of NSCL/P

Junyan Lina,b,c,d, Xiaofeng Lia,b,c,d, Ji Maoa,b,c,d, Xinyu Zhanga,b,c, Tingting Chenga,b,c, Yue Gaoa,b,c, Shu Loua,b,c( ), Dandan Lia,b,c( ), Yongchu Pana,b,c( )
Department of Orthodontics, The Affiliated Stomatological Hospital of Nanjing Medical University, Nanjing, Jiangsu 210029, China
State Key Laboratory Cultivation Base of Research, Prevention and Treatment for Oral Diseases (Nanjing Medical University), Nanjing, Jiangsu 210029, China
Jiangsu Province Engineering Research Center of Stomatological Translational Medicine (Nanjing Medical University), Nanjing, Jiangsu 210029, China

d These authors contributed equally to this work.

Peer review under the responsibility of Chongqing Medical University.

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Genes & Diseases
Volume 13 Issue 4,
July 2026
DOI: 10.1016/j.gendis.2025.101875

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Cite this article:
Lin J, Li X, Mao J, et al. Cis-regulation of FAM49A by a risk variant at 2p24 contributes to the genetic susceptibility of NSCL/P. Genes & Diseases, 2026, 13(4). https://doi.org/10.1016/j.gendis.2025.101875

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Received: 16 July 2025
Published: 30 September 2025
© 2025 The Authors.

This is an open access article under the CC BY license (http://creativecommons.org/licenses/by/4.0/).