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Rapid Communication | Open Access

Tnni3k regulates cardiomyopathy and cardiac conduction disease through Nfatc1 signaling

Shi Ouyanga,b,e,1, Yujuan Niuc,d,1, Le Liua,b, Qiaorong Yia,b, Wuming Qina,b, Hui Caoa,b, Tao Liaoa,b, Rong Xiangf, Yonghe Dingc,d( ), Yun Denga,b,e( )
State Key Laboratory of Developmental Biology of Freshwater Fish, Hunan Normal University, Changsha, Hunan 410081, China
Laboratory of Zebrafish Genetics, College of Life Sciences, Hunan Normal University, Changsha, Hunan 410081, China
The Affiliated Hospital of Qingdao University, Qingdao University, Qingdao, Shandong 266071, China
The Biomedical Sciences Institute of Qingdao University (Qingdao Branch of SJTU Bio-X Institute), Qingdao University, Qingdao, Shandong 266071, China
NHC Key Laboratory of Birth Defect for Research and Prevention, Hunan Provincial Maternal and Child Health Care Hospital, Changsha, Hunan 410028, China
Development of Cell Biology, The School of Life Sciences, Central South University, Changsha, Hunan 410083, China

Peer review under the responsibility of the Genes & Diseases Editorial Office, in alliance with the Association of Chinese Americans in Cancer Research (ACACR, Baltimore, MD, USA).

1 These authors contributed equally to this work.

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Genes & Diseases
Volume 12 Issue 3,
May 2025
Article number: 101464
DOI: 10.1016/j.gendis.2024.101464

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Cite this article:
Ouyang S, Niu Y, Liu L, et al. Tnni3k regulates cardiomyopathy and cardiac conduction disease through Nfatc1 signaling. Genes & Diseases, 2025, 12(3): 101464. https://doi.org/10.1016/j.gendis.2024.101464

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Received: 05 September 2023
Published: 13 November 2024
© 2024 The Authors.

This is an open access article under the CC BY license (http://creativecommons.org/licenses/by/4.0/).